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Article Excerpt
The identification of two cellular receptors that likely contribute to the genesis of hormone-dependent breast cancer points the way to new, highly targeted therapies against the disease, said a team led by scientists at Weill Cornell Medical College in New York City.
The finding also helps explain how daily use of medicines such as aspirin might help keep these breast tumors at bay.
"These two receptors, called EP2 and EP4, form key links in a biochemical pathway that boosts estrogen production in fat and breast cancer cells -- this, in turn, may increase a woman's risk for developing hormone receptor-positive breast cancer. Finding ways to interrupt this pathway in a manner that causes few side effects is the ultimate goal of this research," explains the study's senior author Dr. Andrew Dannenberg, director of the newly established Cancer Center at NewYork-Presbyterian Hospital/Weill Cornell Medical Center, and the Henry R. Erle, M.D.--Roberts Family Professor of Medicine at Weill Cornell Medical College.
The new...
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